Manuel Serrano, a researcher at the Spanish National Tumor Research Center in Madrid, says the body ages because of the accumulation of faulty cells and p53 delays ageing for a similar cause that it prevents tumor. Related StoriesMD Anderson research reveals why chemotherapy medications not effective for many pancreatic tumor patientsNew RNA check of blood platelets can be used to detect area of cancerFDA grants accelerated authorization for Tagrisso to take care of patients with advanced NSCLCThe p53 gene, when it properly is working, ensures that damaged cells destroy themselves , nor divide to cause tumors uncontrollably; if the gene is normally mutated or inactivated the control mechanism does not work.Scientists have known for quite a while that CDKN1C plays a significant function in regulating cell development now. Mutations of the gene have already been previously associated with an ‘overgrowth’ syndrome known as Beckwith-Wiedemann syndrome, which in turn causes large body parts and huge organs and bears an increased risk of tumours. The link with IMAGe is the first time that the gene has been associated with development restriction. We found out a mutation in chromosome 11 that consistently made an appearance atlanta divorce attorneys family member affected by IMAGe Syndrome, clarifies Professor Vilain.